Redox regulation of inflammatory processes
- Autores
- Valacchi, Giuseppe; Cervellati, Carlo; Evelson, Pablo Andrés; Rahman, Irfan
- Año de publicación
- 2016
- Idioma
- inglés
- Tipo de recurso
- artículo
- Estado
- versión publicada
- Descripción
- Oxidative stress mediators such as Reactive Oxygen Species (ROS) and Reactive Nitrogen Species (RNS) can be considered double edge molecules, whenever their role in inflammatory pathways is considered. Indeed, if on one hand ROS/RNS are important inflammatory effectors involved in the invading pathogens clearance and resolving the damage and inducing tissue repair; on the other hand, ROS/RNS can also be inflammatory initiators as a consequence of oxidizing biomolecules such as lipids, proteins and DNA, leading to tissue damage and switching acute to chronic inflammation.
Fil: Valacchi, Giuseppe. Università di Ferrara; Italia. University of North Carolina; Estados Unidos
Fil: Cervellati, Carlo. Università di Ferrara; Italia
Fil: Evelson, Pablo Andrés. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Bioquímica y Medicina Molecular. Universidad de Buenos Aires. Facultad Medicina. Instituto de Bioquímica y Medicina Molecular; Argentina
Fil: Rahman, Irfan. University of Rochester; Estados Unidos - Materia
-
Oxidative Stress
Inflammation
Redox Signaling
Reactive Oxygen Species - Nivel de accesibilidad
- acceso abierto
- Condiciones de uso
- https://creativecommons.org/licenses/by-nc-nd/2.5/ar/
- Repositorio
- Institución
- Consejo Nacional de Investigaciones Científicas y Técnicas
- OAI Identificador
- oai:ri.conicet.gov.ar:11336/47357
Ver los metadatos del registro completo
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Redox regulation of inflammatory processesValacchi, GiuseppeCervellati, CarloEvelson, Pablo AndrésRahman, IrfanOxidative StressInflammationRedox SignalingReactive Oxygen Specieshttps://purl.org/becyt/ford/3.3https://purl.org/becyt/ford/3Oxidative stress mediators such as Reactive Oxygen Species (ROS) and Reactive Nitrogen Species (RNS) can be considered double edge molecules, whenever their role in inflammatory pathways is considered. Indeed, if on one hand ROS/RNS are important inflammatory effectors involved in the invading pathogens clearance and resolving the damage and inducing tissue repair; on the other hand, ROS/RNS can also be inflammatory initiators as a consequence of oxidizing biomolecules such as lipids, proteins and DNA, leading to tissue damage and switching acute to chronic inflammation.Fil: Valacchi, Giuseppe. Università di Ferrara; Italia. University of North Carolina; Estados UnidosFil: Cervellati, Carlo. Università di Ferrara; ItaliaFil: Evelson, Pablo Andrés. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Bioquímica y Medicina Molecular. Universidad de Buenos Aires. Facultad Medicina. Instituto de Bioquímica y Medicina Molecular; ArgentinaFil: Rahman, Irfan. University of Rochester; Estados UnidosPergamon-Elsevier Science Ltd2016-12info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionhttp://purl.org/coar/resource_type/c_6501info:ar-repo/semantics/articuloapplication/pdfapplication/pdfhttp://hdl.handle.net/11336/47357Valacchi, Giuseppe; Cervellati, Carlo; Evelson, Pablo Andrés; Rahman, Irfan; Redox regulation of inflammatory processes; Pergamon-Elsevier Science Ltd; International Journal of Biochemistry and Cellular Biology; 81; Part B; 12-2016; 234-2351357-2725CONICET DigitalCONICETenginfo:eu-repo/semantics/altIdentifier/doi/10.1016/j.biocel.2016.11.002info:eu-repo/semantics/altIdentifier/url/https://www.sciencedirect.com/science/article/pii/S1357272516303338info:eu-repo/semantics/openAccesshttps://creativecommons.org/licenses/by-nc-nd/2.5/ar/reponame:CONICET Digital (CONICET)instname:Consejo Nacional de Investigaciones Científicas y Técnicas2025-09-29T10:28:32Zoai:ri.conicet.gov.ar:11336/47357instacron:CONICETInstitucionalhttp://ri.conicet.gov.ar/Organismo científico-tecnológicoNo correspondehttp://ri.conicet.gov.ar/oai/requestdasensio@conicet.gov.ar; lcarlino@conicet.gov.arArgentinaNo correspondeNo correspondeNo correspondeopendoar:34982025-09-29 10:28:33.163CONICET Digital (CONICET) - Consejo Nacional de Investigaciones Científicas y Técnicasfalse |
dc.title.none.fl_str_mv |
Redox regulation of inflammatory processes |
title |
Redox regulation of inflammatory processes |
spellingShingle |
Redox regulation of inflammatory processes Valacchi, Giuseppe Oxidative Stress Inflammation Redox Signaling Reactive Oxygen Species |
title_short |
Redox regulation of inflammatory processes |
title_full |
Redox regulation of inflammatory processes |
title_fullStr |
Redox regulation of inflammatory processes |
title_full_unstemmed |
Redox regulation of inflammatory processes |
title_sort |
Redox regulation of inflammatory processes |
dc.creator.none.fl_str_mv |
Valacchi, Giuseppe Cervellati, Carlo Evelson, Pablo Andrés Rahman, Irfan |
author |
Valacchi, Giuseppe |
author_facet |
Valacchi, Giuseppe Cervellati, Carlo Evelson, Pablo Andrés Rahman, Irfan |
author_role |
author |
author2 |
Cervellati, Carlo Evelson, Pablo Andrés Rahman, Irfan |
author2_role |
author author author |
dc.subject.none.fl_str_mv |
Oxidative Stress Inflammation Redox Signaling Reactive Oxygen Species |
topic |
Oxidative Stress Inflammation Redox Signaling Reactive Oxygen Species |
purl_subject.fl_str_mv |
https://purl.org/becyt/ford/3.3 https://purl.org/becyt/ford/3 |
dc.description.none.fl_txt_mv |
Oxidative stress mediators such as Reactive Oxygen Species (ROS) and Reactive Nitrogen Species (RNS) can be considered double edge molecules, whenever their role in inflammatory pathways is considered. Indeed, if on one hand ROS/RNS are important inflammatory effectors involved in the invading pathogens clearance and resolving the damage and inducing tissue repair; on the other hand, ROS/RNS can also be inflammatory initiators as a consequence of oxidizing biomolecules such as lipids, proteins and DNA, leading to tissue damage and switching acute to chronic inflammation. Fil: Valacchi, Giuseppe. Università di Ferrara; Italia. University of North Carolina; Estados Unidos Fil: Cervellati, Carlo. Università di Ferrara; Italia Fil: Evelson, Pablo Andrés. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Bioquímica y Medicina Molecular. Universidad de Buenos Aires. Facultad Medicina. Instituto de Bioquímica y Medicina Molecular; Argentina Fil: Rahman, Irfan. University of Rochester; Estados Unidos |
description |
Oxidative stress mediators such as Reactive Oxygen Species (ROS) and Reactive Nitrogen Species (RNS) can be considered double edge molecules, whenever their role in inflammatory pathways is considered. Indeed, if on one hand ROS/RNS are important inflammatory effectors involved in the invading pathogens clearance and resolving the damage and inducing tissue repair; on the other hand, ROS/RNS can also be inflammatory initiators as a consequence of oxidizing biomolecules such as lipids, proteins and DNA, leading to tissue damage and switching acute to chronic inflammation. |
publishDate |
2016 |
dc.date.none.fl_str_mv |
2016-12 |
dc.type.none.fl_str_mv |
info:eu-repo/semantics/article info:eu-repo/semantics/publishedVersion http://purl.org/coar/resource_type/c_6501 info:ar-repo/semantics/articulo |
format |
article |
status_str |
publishedVersion |
dc.identifier.none.fl_str_mv |
http://hdl.handle.net/11336/47357 Valacchi, Giuseppe; Cervellati, Carlo; Evelson, Pablo Andrés; Rahman, Irfan; Redox regulation of inflammatory processes; Pergamon-Elsevier Science Ltd; International Journal of Biochemistry and Cellular Biology; 81; Part B; 12-2016; 234-235 1357-2725 CONICET Digital CONICET |
url |
http://hdl.handle.net/11336/47357 |
identifier_str_mv |
Valacchi, Giuseppe; Cervellati, Carlo; Evelson, Pablo Andrés; Rahman, Irfan; Redox regulation of inflammatory processes; Pergamon-Elsevier Science Ltd; International Journal of Biochemistry and Cellular Biology; 81; Part B; 12-2016; 234-235 1357-2725 CONICET Digital CONICET |
dc.language.none.fl_str_mv |
eng |
language |
eng |
dc.relation.none.fl_str_mv |
info:eu-repo/semantics/altIdentifier/doi/10.1016/j.biocel.2016.11.002 info:eu-repo/semantics/altIdentifier/url/https://www.sciencedirect.com/science/article/pii/S1357272516303338 |
dc.rights.none.fl_str_mv |
info:eu-repo/semantics/openAccess https://creativecommons.org/licenses/by-nc-nd/2.5/ar/ |
eu_rights_str_mv |
openAccess |
rights_invalid_str_mv |
https://creativecommons.org/licenses/by-nc-nd/2.5/ar/ |
dc.format.none.fl_str_mv |
application/pdf application/pdf |
dc.publisher.none.fl_str_mv |
Pergamon-Elsevier Science Ltd |
publisher.none.fl_str_mv |
Pergamon-Elsevier Science Ltd |
dc.source.none.fl_str_mv |
reponame:CONICET Digital (CONICET) instname:Consejo Nacional de Investigaciones Científicas y Técnicas |
reponame_str |
CONICET Digital (CONICET) |
collection |
CONICET Digital (CONICET) |
instname_str |
Consejo Nacional de Investigaciones Científicas y Técnicas |
repository.name.fl_str_mv |
CONICET Digital (CONICET) - Consejo Nacional de Investigaciones Científicas y Técnicas |
repository.mail.fl_str_mv |
dasensio@conicet.gov.ar; lcarlino@conicet.gov.ar |
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1844614289193697280 |
score |
13.070432 |