Redox regulation of inflammatory processes

Autores
Valacchi, Giuseppe; Cervellati, Carlo; Evelson, Pablo Andrés; Rahman, Irfan
Año de publicación
2016
Idioma
inglés
Tipo de recurso
artículo
Estado
versión publicada
Descripción
Oxidative stress mediators such as Reactive Oxygen Species (ROS) and Reactive Nitrogen Species (RNS) can be considered double edge molecules, whenever their role in inflammatory pathways is considered. Indeed, if on one hand ROS/RNS are important inflammatory effectors involved in the invading pathogens clearance and resolving the damage and inducing tissue repair; on the other hand, ROS/RNS can also be inflammatory initiators as a consequence of oxidizing biomolecules such as lipids, proteins and DNA, leading to tissue damage and switching acute to chronic inflammation.
Fil: Valacchi, Giuseppe. Università di Ferrara; Italia. University of North Carolina; Estados Unidos
Fil: Cervellati, Carlo. Università di Ferrara; Italia
Fil: Evelson, Pablo Andrés. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Bioquímica y Medicina Molecular. Universidad de Buenos Aires. Facultad Medicina. Instituto de Bioquímica y Medicina Molecular; Argentina
Fil: Rahman, Irfan. University of Rochester; Estados Unidos
Materia
Oxidative Stress
Inflammation
Redox Signaling
Reactive Oxygen Species
Nivel de accesibilidad
acceso abierto
Condiciones de uso
https://creativecommons.org/licenses/by-nc-nd/2.5/ar/
Repositorio
CONICET Digital (CONICET)
Institución
Consejo Nacional de Investigaciones Científicas y Técnicas
OAI Identificador
oai:ri.conicet.gov.ar:11336/47357
Acceder
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network_name_str CONICET Digital (CONICET)
spelling Redox regulation of inflammatory processesValacchi, GiuseppeCervellati, CarloEvelson, Pablo AndrésRahman, IrfanOxidative StressInflammationRedox SignalingReactive Oxygen Specieshttps://purl.org/becyt/ford/3.3https://purl.org/becyt/ford/3Oxidative stress mediators such as Reactive Oxygen Species (ROS) and Reactive Nitrogen Species (RNS) can be considered double edge molecules, whenever their role in inflammatory pathways is considered. Indeed, if on one hand ROS/RNS are important inflammatory effectors involved in the invading pathogens clearance and resolving the damage and inducing tissue repair; on the other hand, ROS/RNS can also be inflammatory initiators as a consequence of oxidizing biomolecules such as lipids, proteins and DNA, leading to tissue damage and switching acute to chronic inflammation.Fil: Valacchi, Giuseppe. Università di Ferrara; Italia. University of North Carolina; Estados UnidosFil: Cervellati, Carlo. Università di Ferrara; ItaliaFil: Evelson, Pablo Andrés. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Bioquímica y Medicina Molecular. Universidad de Buenos Aires. Facultad Medicina. Instituto de Bioquímica y Medicina Molecular; ArgentinaFil: Rahman, Irfan. University of Rochester; Estados UnidosPergamon-Elsevier Science Ltd2016-12info:eu-repo/semantics/articleinfo:eu-repo/semantics/publishedVersionhttp://purl.org/coar/resource_type/c_6501info:ar-repo/semantics/articuloapplication/pdfapplication/pdfhttp://hdl.handle.net/11336/47357Valacchi, Giuseppe; Cervellati, Carlo; Evelson, Pablo Andrés; Rahman, Irfan; Redox regulation of inflammatory processes; Pergamon-Elsevier Science Ltd; International Journal of Biochemistry and Cellular Biology; 81; Part B; 12-2016; 234-2351357-2725CONICET DigitalCONICETenginfo:eu-repo/semantics/altIdentifier/doi/10.1016/j.biocel.2016.11.002info:eu-repo/semantics/altIdentifier/url/https://www.sciencedirect.com/science/article/pii/S1357272516303338info:eu-repo/semantics/openAccesshttps://creativecommons.org/licenses/by-nc-nd/2.5/ar/reponame:CONICET Digital (CONICET)instname:Consejo Nacional de Investigaciones Científicas y Técnicas2025-10-15T15:25:36Zoai:ri.conicet.gov.ar:11336/47357instacron:CONICETInstitucionalhttp://ri.conicet.gov.ar/Organismo científico-tecnológicoNo correspondehttp://ri.conicet.gov.ar/oai/requestdasensio@conicet.gov.ar; lcarlino@conicet.gov.arArgentinaNo correspondeNo correspondeNo correspondeopendoar:34982025-10-15 15:25:36.492CONICET Digital (CONICET) - Consejo Nacional de Investigaciones Científicas y Técnicasfalse
dc.title.none.fl_str_mv Redox regulation of inflammatory processes
title Redox regulation of inflammatory processes
spellingShingle Redox regulation of inflammatory processes
Valacchi, Giuseppe
Oxidative Stress
Inflammation
Redox Signaling
Reactive Oxygen Species
title_short Redox regulation of inflammatory processes
title_full Redox regulation of inflammatory processes
title_fullStr Redox regulation of inflammatory processes
title_full_unstemmed Redox regulation of inflammatory processes
title_sort Redox regulation of inflammatory processes
dc.creator.none.fl_str_mv Valacchi, Giuseppe
Cervellati, Carlo
Evelson, Pablo Andrés
Rahman, Irfan
author Valacchi, Giuseppe
author_facet Valacchi, Giuseppe
Cervellati, Carlo
Evelson, Pablo Andrés
Rahman, Irfan
author_role author
author2 Cervellati, Carlo
Evelson, Pablo Andrés
Rahman, Irfan
author2_role author
author
author
dc.subject.none.fl_str_mv Oxidative Stress
Inflammation
Redox Signaling
Reactive Oxygen Species
topic Oxidative Stress
Inflammation
Redox Signaling
Reactive Oxygen Species
purl_subject.fl_str_mv https://purl.org/becyt/ford/3.3
https://purl.org/becyt/ford/3
dc.description.none.fl_txt_mv Oxidative stress mediators such as Reactive Oxygen Species (ROS) and Reactive Nitrogen Species (RNS) can be considered double edge molecules, whenever their role in inflammatory pathways is considered. Indeed, if on one hand ROS/RNS are important inflammatory effectors involved in the invading pathogens clearance and resolving the damage and inducing tissue repair; on the other hand, ROS/RNS can also be inflammatory initiators as a consequence of oxidizing biomolecules such as lipids, proteins and DNA, leading to tissue damage and switching acute to chronic inflammation.
Fil: Valacchi, Giuseppe. Università di Ferrara; Italia. University of North Carolina; Estados Unidos
Fil: Cervellati, Carlo. Università di Ferrara; Italia
Fil: Evelson, Pablo Andrés. Consejo Nacional de Investigaciones Científicas y Técnicas. Oficina de Coordinación Administrativa Houssay. Instituto de Bioquímica y Medicina Molecular. Universidad de Buenos Aires. Facultad Medicina. Instituto de Bioquímica y Medicina Molecular; Argentina
Fil: Rahman, Irfan. University of Rochester; Estados Unidos
description Oxidative stress mediators such as Reactive Oxygen Species (ROS) and Reactive Nitrogen Species (RNS) can be considered double edge molecules, whenever their role in inflammatory pathways is considered. Indeed, if on one hand ROS/RNS are important inflammatory effectors involved in the invading pathogens clearance and resolving the damage and inducing tissue repair; on the other hand, ROS/RNS can also be inflammatory initiators as a consequence of oxidizing biomolecules such as lipids, proteins and DNA, leading to tissue damage and switching acute to chronic inflammation.
publishDate 2016
dc.date.none.fl_str_mv 2016-12
dc.type.none.fl_str_mv info:eu-repo/semantics/article
info:eu-repo/semantics/publishedVersion
http://purl.org/coar/resource_type/c_6501
info:ar-repo/semantics/articulo
format article
status_str publishedVersion
dc.identifier.none.fl_str_mv http://hdl.handle.net/11336/47357
Valacchi, Giuseppe; Cervellati, Carlo; Evelson, Pablo Andrés; Rahman, Irfan; Redox regulation of inflammatory processes; Pergamon-Elsevier Science Ltd; International Journal of Biochemistry and Cellular Biology; 81; Part B; 12-2016; 234-235
1357-2725
CONICET Digital
CONICET
url http://hdl.handle.net/11336/47357
identifier_str_mv Valacchi, Giuseppe; Cervellati, Carlo; Evelson, Pablo Andrés; Rahman, Irfan; Redox regulation of inflammatory processes; Pergamon-Elsevier Science Ltd; International Journal of Biochemistry and Cellular Biology; 81; Part B; 12-2016; 234-235
1357-2725
CONICET Digital
CONICET
dc.language.none.fl_str_mv eng
language eng
dc.relation.none.fl_str_mv info:eu-repo/semantics/altIdentifier/doi/10.1016/j.biocel.2016.11.002
info:eu-repo/semantics/altIdentifier/url/https://www.sciencedirect.com/science/article/pii/S1357272516303338
dc.rights.none.fl_str_mv info:eu-repo/semantics/openAccess
https://creativecommons.org/licenses/by-nc-nd/2.5/ar/
eu_rights_str_mv openAccess
rights_invalid_str_mv https://creativecommons.org/licenses/by-nc-nd/2.5/ar/
dc.format.none.fl_str_mv application/pdf
application/pdf
dc.publisher.none.fl_str_mv Pergamon-Elsevier Science Ltd
publisher.none.fl_str_mv Pergamon-Elsevier Science Ltd
dc.source.none.fl_str_mv reponame:CONICET Digital (CONICET)
instname:Consejo Nacional de Investigaciones Científicas y Técnicas
reponame_str CONICET Digital (CONICET)
collection CONICET Digital (CONICET)
instname_str Consejo Nacional de Investigaciones Científicas y Técnicas
repository.name.fl_str_mv CONICET Digital (CONICET) - Consejo Nacional de Investigaciones Científicas y Técnicas
repository.mail.fl_str_mv dasensio@conicet.gov.ar; lcarlino@conicet.gov.ar
_version_ 1846083400392245248
score 13.22299

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